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Upregulation and translocation of nitric oxide synthase (NOS) induce activation of Ca2+-activated K+ channels (KCa) in smooth muscle cells.
Gap junctional communication between vascular smooth muscle cells (SMC) results in changes in vascular tone and helps maintain blood pressure. In this study, we observed that stimulation of SMC with nitric oxide (NO) upregulated the synthesis of cyclic guanosine 3',5'-monophosphate ( https://cse.google.nl/url?q=https://rimerloso.weebly.com

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